The Pine tree has remarkable properties and promises help for present needs.
Historically, pine has been used for many different aliments and especially lung issues. Presently, Pine needle tea is being reputed as helpful for Spike proteins and C—d. In fact Suramin, which is considered the antidote to C—d is derived from Pine.
Historical Uses of Pine
In the book, Back to Eden, Dr. Jethro Kloss states,
“White pine is a very old reliable remedy for chest ailments such as bronchitis, coughs, colds, croup, and influenza. It is excellent for use in tonsillitis, laryngitis, and sore throats. It will stop coughing and help to expel phlegm from the throats. It is even better when combined with wild cherry bark or spikenard.
It has also been found useful in rheumatism, kidney trouble, and scurvy. Combined with uva ursi, marshmallow, and poplar bark i is excellent for diabetes. The American Indians used the pine tree for food as well as medicine by making bread from the ground up bark. This was their remedy for kidney, throat, and lung afflictions, especially sore throat and tonsillitis.” [1]
Modern uses
The the website for Bioregulatory Medicine Institute, Dr. James Odell writes about Eastern White Pine. He states,
“Several compounds found in pine needles have been identified as a potential antidote to the current spike protein contagion resulting from the chimeric SARS-CoV-2 coronavirus, and the potential pathogenic transmission of the spike protein from the experimental mRNA vaccines.
Eastern white pine tree needles (Pinus Strobus) contain many beneficial constituents useful for the prevention of colds and flu such as Alpha-Pinene, Beta-Pinene, Beta-Phellandrene, D-Limonene, Germacrene D, 3-Carene, Caryophyllene, vitamin A, and vitamin C. Eastern white pine needles also contain shikimic acid, the same molecule found in star anise herb used historically in Traditional Chinese Medicine to treat plagues and respiratory illness.”[2]
Suramin, C—d, and Spike Proteins
Dr. Odell continues, “Suramin is strongly inhibitory to the replication of the coronavirus spike protein. It also has an inhibitory effect against components of the coagulation cascade and the inappropriate replication and modification of RNA and DNA. Excessive coagulation causes blood clots, mini-clots, strokes, and unusually heavy menstrual cycles, which are now being daily reported to VAERS post-vaccination. Recent research has shown it to be an in vitro inhibitor of SARS-CoV-2 infection in cell culture by preventing viral entry. Research shows this to be due to its potent inhibition of SARS-CoV-2 RNA-dependent RNA polymerase (RdRp), an essential viral lifecycle enzyme.[2]
Suramin originally is derived from trypan blue derived from toluene a derivative of pine oil.
When suramin was introduced for the treatment of African sleeping sickness in 1922, it was one of the first anti-infective agents that had been developed in a medicinal chemistry program. . . . . Pine needle oil was used as a botanical source for trypan blue. Sleeping sickness (also known as human African trypanosomiasis was at the forefront of research at that time, not a neglected disease as it is today, and the development of suramin was a breakthrough for the emerging field of chemotherapy.
Trypan blue is so-called because it can kill trypanosomes, the parasites that cause sleeping sickness. Trypan blue is derived from toluidine, that is, any of several isomeric bases, C14H16N2, derived from toluene. Trypan blue is also known as diamine blue and Niagara blue. Toluene is also a derivative of pine oil. The compound was first isolated in 1837 through a distillation of pine oil by the Polish chemist Filip Walter, who named it rétinnaphte.[2]
Notes:
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Amy Willis M.H., CN
Resources:
1 Back to Eden, Jethro Kloss, pp 223-224.
2 Eastern White Pine Tree Needles: A Natural Source of Anti-Infective Compounds (biologicalmedicineinstitute.
About the Author
Amy Willis M.H.